A splice variant of the ITF-2 transcript encodes a transcription factor that inhibits MyoD activity.
Skerjanc, I S; Truong, J; Filion, P; McBurney, M W.
J Biol Chem
; 271(7): 3555-61, 1996 Feb 16.
Artigo em Inglês | MEDLINE | ID: mdl-8631961
Low Six4 and Six5 gene dosage improves dystrophic phenotype and prolongs life span of mdx mice.
MyoD targets TAF3/TRF3 to activate myogenin transcription.
RNA Decay Factor UPF1 Promotes Protein Decay: A Hidden Talent.
The RNA Surveillance Factor UPF1 Represses Myogenesis via Its E3 Ubiquitin Ligase Activity.
eRNAs promote transcription by establishing chromatin accessibility at defined genomic loci.
Oscillations of MyoD and Hes1 proteins regulate the maintenance of activated muscle stem cells.
Human skeletal muscle-derived CD133(+) cells form functional satellite cells after intramuscular transplantation in immunodeficient host mice.
CLOCK and BMAL1 regulate MyoD and are necessary for maintenance of skeletal muscle phenotype and function.
Genome-wide association between Six4, MyoD, and the histone demethylase Utx during myogenesis.
MyoD phosphorylation on multiple C terminal sites regulates myogenic conversion activity.