Relative resistance to HIV-1 infection of CD4 lymphocytes from persons who remain uninfected despite multiple high-risk sexual exposure.
Paxton, W A; Martin, S R; Tse, D; O'Brien, T R; Skurnick, J; VanDevanter, N L; Padian, N; Braun, J F; Kotler, D P; Wolinsky, S M; Koup, R A.
; 2(4): 412-7, 1996 Apr.
Artigo em Inglês | MEDLINE | ID: mdl-8597950
Multiple Origins of Virus Persistence during Natural Control of HIV Infection.
T cell susceptibility to HIV influences outcome of opportunistic infections.
An integrated overview of HIV-1 latency.
Replication-competent noninduced proviruses in the latent reservoir increase barrier to HIV-1 cure.
Membrane metalloprotease TRABD2A restricts HIV-1 progeny production in resting CD4<sup>+</sup> T cells by degrading viral Gag polyprotein.
Reduced Potency and Incomplete Neutralization of Broadly Neutralizing Antibodies against Cell-to-Cell Transmission of HIV-1 with Transmitted Founder Envs.
Hunting Down the HIV-1 Reservoir: A Starring Role for Antibodies?
HIV Controllers Exhibit Enhanced Frequencies of Major Histocompatibility Complex Class II Tetramer<sup>+</sup> Gag-Specific CD4<sup>+</sup> T Cells in Chronic Clade C HIV-1 Infection.
TCR clonotypes modulate the protective effect of HLA class I molecules in HIV-1 infection.
Expanded cellular clones carrying replication-competent HIV-1 persist, wax, and wane.