Relative resistance to HIV-1 infection of CD4 lymphocytes from persons who remain uninfected despite multiple high-risk sexual exposure.
Paxton, W A; Martin, S R; Tse, D; O'Brien, T R; Skurnick, J; VanDevanter, N L; Padian, N; Braun, J F; Kotler, D P; Wolinsky, S M; Koup, R A.
; 2(4): 412-7, 1996 Apr.
Artigo em Inglês | MEDLINE | ID: mdl-8597950
The Immune Response to Mycobacterium tuberculosis in HIV-1-Coinfected Persons.
Resistance of HIV-infected macrophages to CD8<sup>+</sup> T lymphocyte-mediated killing drives activation of the immune system.
Tissue memory CD4+ T cells expressing IL-7 receptor-alpha (CD127) preferentially support latent HIV-1 infection.
An integrated overview of HIV-1 latency.
Robust and persistent reactivation of SIV and HIV by N-803 and depletion of CD8<sup>+</sup> cells.
T cell susceptibility to HIV influences outcome of opportunistic infections.
Chromatin maturation of the HIV-1 provirus in primary resting CD4+ T cells.
Multiple Origins of Virus Persistence during Natural Control of HIV Infection.
Replication-competent noninduced proviruses in the latent reservoir increase barrier to HIV-1 cure.
HIV-1-induced cytokines deplete homeostatic innate lymphoid cells and expand TCF7-dependent memory NK cells.