The Vin-1 gene, identified by provirus insertional mutagenesis, is the cyclin D2.
Hanna, Z; Jankowski, M; Tremblay, P; Jiang, X; Milatovich, A; Francke, U; Jolicoeur, P.
; 8(6): 1661-6, 1993 Jun.
Artigo em Inglês | MEDLINE | ID: mdl-8502486
Widespread shortening of 3'UTRs by alternative cleavage and polyadenylation activates oncogenes in cancer cells.
TACI expression is associated with a mature bone marrow plasma cell signature and C-MAF overexpression in human myeloma cell lines.
Therapeutic microRNA delivery suppresses tumorigenesis in a murine liver cancer model.
miR-26a regulates mouse hepatocyte proliferation via directly targeting the 3' untranslated region of CCND2 and CCNE2.
Modified STAP conditions facilitate bivalent fate decision between pluripotency and apoptosis in Jurkat T-lymphocytes.
Asymmetric inheritance of Cyclin D2 maintains proliferative neural stem/progenitor cells: a critical event in brain development and evolution.
Systematic identification of factors for provirus silencing in embryonic stem cells.
Epstein-Barr Virus Nuclear Antigen 3C Facilitates Cell Proliferation by Regulating Cyclin D2.
Enhancer methylation dynamics contribute to cancer plasticity and patient mortality.