Myc-induced nuclear antigen constrains a latent intestinal epithelial cell-intrinsic anthelmintic pathway.
Pillai, Meenu R; Mihi, Belgacem; Ishiwata, Kenji; Nakamura, Kiminori; Sakuragi, Naoya; Finkelstein, David B; McGargill, Maureen A; Nakayama, Toshinori; Ayabe, Tokiyoshi; Coleman, Mathew L; Bix, Mark.
; 14(2): e0211244, 2019.
Artigo em Inglês | MEDLINE | ID: mdl-30807587
Distinct DC subsets regulate adaptive Th1 and 2 responses during Trichuris muris infection.
Chronic Trichuris muris infection alters hematopoiesis and causes IFN-Î³-expressing T-cell accumulation in the mouse bone marrow.
Increased susceptibility to oral Trichuris muris infection in the specific absence of CXCR5<sup>+</sup> CD11c<sup>+</sup> cells.
Loss of the TGFß-activating integrin αvß8 on dendritic cells protects mice from chronic intestinal parasitic infection via control of type 2 immunity.
IL25 elicits a multipotent progenitor cell population that promotes T(H)2 cytokine responses.
Acute infection with the intestinal parasite Trichuris muris has long-term consequences on mucosal mast cell homeostasis and epithelial integrity.
Rapid dendritic cell mobilization to the large intestinal epithelium is associated with resistance to Trichuris muris infection.
Dendritic-cell expression of Ship1 regulates Th2 immunity to helminth infection in mice.
The NLRP3 Inflammasome Suppresses Protective Immunity to Gastrointestinal Helminth Infection.
The role of sex hormones in the development of Th2 immunity in a gender-biased model of Trichuris muris infection.