An X-linked Myh11-CreER<sup>T2</sup> mouse line resulting from Y to X chromosome-translocation of the Cre allele.
Liao, Mingmei; Zhou, Junmei; Wang, Fen; Ali, Yasmin H; Chan, Kelvin L; Zou, Fei; Offermanns, Stefan; Jiang, Zhisheng; Jiang, Zhihua.
; 55(9)2017 09.
Artigo em Inglês | MEDLINE | ID: mdl-28845554
Cardiac-Specific Cre Induces Age-Dependent Dilated Cardiomyopathy (DCM) in Mice.
Independent evolution of striated muscles in cnidarians and bilaterians.
Myosin VI dimerization triggers an unfolding of a three-helix bundle in order to extend its reach.
Prolonged Cre expression driven by the α-myosin heavy chain promoter can be cardiotoxic.
Mutations in embryonic myosin heavy chain (MYH3) cause Freeman-Sheldon syndrome and Sheldon-Hall syndrome.
Germ line activation of the Tie2 and SMMHC promoters causes noncell-specific deletion of floxed alleles.
Myosin VI rewrites the rules for myosin motors.
<i>Drosophila</i> model of myosin myopathy rescued by overexpression of a TRIM-protein family member.
Investigations of human myosin VI targeting using optogenetically controlled cargo loading.
Overexpression of smooth muscle myosin heavy chain leads to activation of the unfolded protein response and autophagic turnover of thick filament-associated proteins in vascular smooth muscle cells.