Dual role of MUC1 mucin in kidney ischemia-reperfusion injury: Nephroprotector in early phase, but pro-fibrotic in late phase.
Gibier, Jean-Baptiste; Hémon, Brigitte; Fanchon, Mélanie; Gaudelot, Kelly; Pottier, Nicolas; Ringot, Bélinda; Van Seuningen, Isabelle; Glowacki, François; Cauffiez, Christelle; Blum, David; Copin, Marie-Christine; Perrais, Michaël; Gnemmi, Viviane.
Biochim Biophys Acta Mol Basis Dis
; 1863(6): 1336-1349, 2017 06.
Artigo em Inglês | MEDLINE | ID: mdl-28366875
[The role of pharmacological preconditioning in renal ischemic and reperfusion injury].
The examination of protective effects of gallic acid against damage of oxidative stress during induced-experimental renal ischemia-reperfusion in experiment.
Bone Marrow-Derived Mononuclear Cell Therapy Accelerates Renal Ischemia-Reperfusion Injury Recovery by Modulating Inflammatory, Antioxidant and Apoptotic Related Molecules.
Ouabain Contributes to Kidney Damage in a Rat Model of Renal Ischemia-Reperfusion Injury.
Mammalian STE20-Like Kinase 1 Deletion Alleviates Renal Ischaemia-Reperfusion Injury via Modulating Mitophagy and the AMPK-YAP Signalling Pathway.
Aquaporin-3 deletion in mice results in renal collecting duct abnormalities and worsens ischemia-reperfusion injury.
miR-21 contributes to renal protection by targeting prolyl hydroxylase domain protein 2 in delayed ischaemic preconditioning.
Heparanase regulates the M1 polarization of renal macrophages and their crosstalk with renal epithelial tubular cells after ischemia/reperfusion injury.
GPR4 knockout improves renal ischemia-reperfusion injury and inhibits apoptosis via suppressing the expression of CHOP.