Differential paralog divergence modulates genome evolution across yeast species.
Sanchez, Monica R; Miller, Aaron W; Liachko, Ivan; Sunshine, Anna B; Lynch, Bryony; Huang, Mei; Alcantara, Erica; DeSevo, Christopher G; Pai, Dave A; Tucker, Cheryl M; Hoang, Margaret L; Dunham, Maitreya J.
; 13(2): e1006585, 2017 02.
Artigo em Inglês | MEDLINE | ID: mdl-28196070
The Complete Pathway for Thiosulfate Utilization in Saccharomyces cerevisiae.
Dysplastic spondylolysis is caused by mutations in the diastrophic dysplasia sulfate transporter gene.
The STAS domain of mammalian SLC26A5 prestin harbours an anion-binding site.
Claudin expression during early postnatal development of the murine cochlea.
SLC26A2 disease spectrum in Sweden - high frequency of recessive multiple epiphyseal dysplasia (rMED).
Sul1 and Sul2 sulfate transceptors signal to protein kinase A upon exit of sulfur starvation.
Comprehensive Analysis of the SUL1 Promoter of Saccharomyces cerevisiae.
Molecular dynamics simulations of the STAS domains of rat prestin and human pendrin reveal conformational motions in conserved flexible regions.
The V499G/Y501H mutation impairs fast motor kinetics of prestin and has significance for defining functional independence of individual prestin subunits.
Origin-dependent inverted-repeat amplification: a replication-based model for generating palindromic amplicons.