Hepatitis C virus infection triggers a tumor-like glutamine metabolism.
Lévy, Pierre L; Duponchel, Sarah; Eischeid, Hannah; Molle, Jennifer; Michelet, Maud; Diserens, Gaëlle; Vermathen, Martina; Vermathen, Peter; Dufour, Jean-Francois; Dienes, Hans-Peter; Steffen, Hans-Michael; Odenthal, Margarete; Zoulim, Fabien; Bartosch, Birke.
; 65(3): 789-803, 2017 03.
Artigo em Inglês | MEDLINE | ID: mdl-27863447
MicroRNA-155 and microRNA-196b: promising biomarkers in hepatitis C virus infection?
A cell culture system for distinguishing hepatitis C viruses with and without liver cancer-related mutations in the viral core gene.
Hepatitis C virus and proprotein convertase subtilisin/kexin type 9: a detrimental interaction to increase viral infectivity and disrupt lipid metabolism.
Molecular pathways: hepatitis C virus, CXCL10, and the inflammatory road to liver cancer.
Genetic variants of XRCC1 and risk of hepatocellular carcinoma in chronic hepatitis C patients.
Hepatitis C viral proteins perturb metabolic liver zonation.
Hepatitis C Virus-Induced Upregulation of MicroRNA miR-146a-5p in Hepatocytes Promotes Viral Infection and Deregulates Metabolic Pathways Associated with Liver Disease Pathogenesis.
RNA triphosphatase DUSP11 enables exonuclease XRN-mediated restriction of hepatitis C virus.
Combined Analysis of Metabolomes, Proteomes, and Transcriptomes of Hepatitis C Virus-Infected Cells and Liver to Identify Pathways Associated With Disease Development.
Genetic variants underlying vitamin D metabolism and VDR-TGFß-1-SMAD3 interaction may impact on HCV progression: a study based on dbGaP data from the HALT-C study.