Human mutations affect the epigenetic/bookmarking function of HNF1B.
Lerner, Jonathan; Bagattin, Alessia; Verdeguer, Francisco; Makinistoglu, Munevver P; Garbay, Serge; Felix, Tristan; Heidet, Laurence; Pontoglio, Marco.
Nucleic Acids Res
; 44(17): 8097-111, 2016 09 30.
Artigo em Inglês | MEDLINE | ID: mdl-27229139
Hepatocyte Nuclear Factor-1<i>ß</i> Controls Mitochondrial Respiration in Renal Tubular Cells.
Transcription Factor Hepatocyte Nuclear Factor-1ß Regulates Renal Cholesterol Metabolism.
Retinoic acid-induced expression of <i>Hnf1b</i> and <i>Fzd4</i> is required for pancreas development in <i>Xenopus laevis</i>.
Genomic integration of ERRÎ³-HNF1ß regulates renal bioenergetics and prevents chronic kidney disease.
Obesity-induced overexpression of miR-802 impairs glucose metabolism through silencing of Hnf1b.
Transcription Factor Hepatocyte Nuclear Factor-1ß (HNF-1ß) Regulates MicroRNA-200 Expression through a Long Noncoding RNA.
All-trans-retinoic Acid Increases SLC26A3 DRA (Down-regulated in Adenoma) Expression in Intestinal Epithelial Cells via HNF-1ß.
Notch is required for the formation of all nephron segments and primes nephron progenitors for differentiation.
Hepatocyte Nuclear Factor-1<i>ß</i> Regulates Urinary Concentration and Response to Hypertonicity.
Hnf1b controls pancreas morphogenesis and the generation of Ngn3+ endocrine progenitors.