Oncogenic BRAF-Mediated Melanoma Cell Invasion.
Lu, Hezhe; Liu, Shujing; Zhang, Gao; Kwong, Lawrence N; Zhu, Yueyao; Miller, John P; Hu, Yi; Zhong, Wenqun; Zeng, Jingwen; Wu, Lawrence; Krepler, Clemens; Sproesser, Katrin; Xiao, Min; Xu, Wei; Karakousis, Giorgos C; Schuchter, Lynn M; Field, Jeffery; Zhang, Paul J; Herlyn, Meenhard; Xu, Xiaowei; Guo, Wei.
; 15(9): 2012-24, 2016 05 31.
Artigo em Inglês | MEDLINE | ID: mdl-27210749
Ultraviolet radiation accelerates BRAF-driven melanomagenesis by targeting TP53.
Massive reshaping of genome-nuclear lamina interactions during oncogene-induced senescence.
Kinase-dead BRAF and oncogenic RAS cooperate to drive tumor progression through CRAF.
The BRAF pseudogene functions as a competitive endogenous RNA and induces lymphoma in vivo.
Oncogenic BRAF induces senescence and apoptosis through pathways mediated by the secreted protein IGFBP7.
Melanoma: from mutations to medicine.
A landscape of driver mutations in melanoma.
The effect of a widespread cancer-causing mutation on the inactive to active dynamics of the B-Raf kinase.
Sequence-Specific Covalent Capture Coupled with High-Contrast Nanopore Detection of a Disease-Derived Nucleic Acid Sequence.
Knock-in(g) RAF for a loop.