APOL1-associated glomerular disease among African-American children: a collaboration of the Chronic Kidney Disease in Children (CKiD) and Nephrotic Syndrome Study Network (NEPTUNE) cohorts.
Ng, Derek K; Robertson, Catherine C; Woroniecki, Robert P; Limou, Sophie; Gillies, Christopher E; Reidy, Kimberly J; Winkler, Cheryl A; Hingorani, Sangeeta; Gibson, Keisha L; Hjorten, Rebecca; Sethna, Christine B; Kopp, Jeffrey B; Moxey-Mims, Marva; Furth, Susan L; Warady, Bradley A; Kretzler, Matthias; Sedor, John R; Kaskel, Frederick J; Sampson, Matthew G.
Nephrol Dial Transplant
; 32(6): 983-990, 2017 Jun 01.
Artigo em Inglês | MEDLINE | ID: mdl-27190333
Genetic Basis of Health Disparity in Childhood Nephrotic Syndrome.
Identification of genetic causes for sporadic steroid-resistant nephrotic syndrome in adults.
Advances in molecular diagnosis and therapeutics in nephrotic syndrome and focal and segmental glomerulosclerosis.
One Actor, Many Roles: Histopathologies Associated With APOL1 Genetic Variants.
Protective association between JC polyoma viruria and kidney disease.
HLA-DQA1 and APOL1 as Risk Loci for Childhood-Onset Steroid-Sensitive and Steroid-Resistant Nephrotic Syndrome.
Integrative Genomics Identifies Novel Associations with APOL1 Risk Genotypes in Black NEPTUNE Subjects.
<i>UBD</i> modifies <i>APOL1</i>-induced kidney disease risk.
Worldwide Frequencies of APOL1 Renal Risk Variants.
Genome-wide association studies suggest that APOL1-environment interactions more likely trigger kidney disease in African Americans with nondiabetic nephropathy than strong APOL1-second gene interactions.