Evidence for human transmission of amyloid-ß pathology and cerebral amyloid angiopathy.
Jaunmuktane, Zane; Mead, Simon; Ellis, Matthew; Wadsworth, Jonathan D F; Nicoll, Andrew J; Kenny, Joanna; Launchbury, Francesca; Linehan, Jacqueline; Richard-Loendt, Angela; Walker, A Sarah; Rudge, Peter; Collinge, John; Brandner, Sebastian.
; 525(7568): 247-50, 2015 Sep 10.
Artigo em Inglês | MEDLINE | ID: mdl-26354483
Dura mater is a potential source of Aß seeds.
Seeds of neuroendocrine doubt.
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Loss of clusterin shifts amyloid deposition to the cerebrovasculature via disruption of perivascular drainage pathways.
Is Alzheimer's disease amyloidosis the result of a repair mechanism gone astray?
Early-onset formation of parenchymal plaque amyloid abrogates cerebral microvascular amyloid accumulation in transgenic mice.
Creutzfeldt-Jakob disease, cerebral amyloid angiopathy and Aß-related angiitis with neuropsychiatric manifestations.
Animal models of cerebral amyloid angiopathy.
Neurodegeneration: Amyloid-ß pathology induced in humans.