FAS Inactivation Releases Unconventional Germinal Center B Cells that Escape Antigen Control and Drive IgE and Autoantibody Production.
Butt, Danyal; Chan, Tyani D; Bourne, Katherine; Hermes, Jana R; Nguyen, Akira; Statham, Aaron; O'Reilly, Lorraine A; Strasser, Andreas; Price, Susan; Schofield, Peter; Christ, Daniel; Basten, Antony; Ma, Cindy S; Tangye, Stuart G; Phan, Tri Giang; Rao, V Koneti; Brink, Robert.
; 42(5): 890-902, 2015 May 19.
Artigo em Inglês | MEDLINE | ID: mdl-25979420
Germinal center quality control: death by Fas.
Identification of a novel type 2 innate immunocyte with the ability to enhance IgE production.
Disturbed B-lymphocyte selection in autoimmune lymphoproliferative syndrome.
The many roles of FAS receptor signaling in the immune system.
Defective survival of naive CD8+ T lymphocytes in the absence of the beta3 regulatory subunit of voltage-gated calcium channels.
Fas receptor expression in germinal-center B cells is essential for T and B lymphocyte homeostasis.
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Dendritic Cells Regulate Extrafollicular Autoreactive B Cells via T Cells Expressing Fas and Fas Ligand.