Tracking of intertissue migration reveals the origins of tumor-infiltrating monocytes.
Shand, Francis H W; Ueha, Satoshi; Otsuji, Mikiya; Koid, Suang Suang; Shichino, Shigeyuki; Tsukui, Tatsuya; Kosugi-Kanaya, Mizuha; Abe, Jun; Tomura, Michio; Ziogas, James; Matsushima, Kouji.
Proc Natl Acad Sci U S A
; 111(21): 7771-6, 2014 May 27.
Artigo em Inglês | MEDLINE | ID: mdl-24825888
Wild Mouse Gut Microbiota Promotes Host Fitness and Improves Disease Resistance.
Î³Î´ T Cells Support Pancreatic Oncogenesis by Restraining αß T Cell Activation.
Regulation of plasma-cell development.
CXCL12 secretion by bone marrow stromal cells is dependent on cell contact and mediated by connexin-43 and connexin-45 gap junctions.
A Distinct Gene Module for Dysfunction Uncoupled from Activation in Tumor-Infiltrating T Cells.
The calcium-activated nonselective cation channel TRPM4 is essential for the migration but not the maturation of dendritic cells.
Correlation between Th17 cells and tumor microenvironment.
Immature monocytes acquire antigens from other cells in the bone marrow and present them to T cells after maturing in the periphery.
Mobilization and margination of bone marrow Gr-1high monocytes during subclinical endotoxemia predisposes the lungs toward acute injury.
C5aR expression in a novel GFP reporter gene knockin mouse: implications for the mechanism of action of C5aR signaling in T cell immunity.