Hyperactive mariner transposons are created by mutations that disrupt allosterism and increase the rate of transposon end synapsis.
Liu, Danxu; Chalmers, Ronald.
Nucleic Acids Res
; 42(4): 2637-45, 2014 Feb.
Artigo em Inglês | MEDLINE | ID: mdl-24319144
Molecular architecture of the Mos1 paired-end complex: the structural basis of DNA transposition in a eukaryote.
Structural role of the flanking DNA in mariner transposon excision.
Target capture during Mos1 transposition.
Molecular Mechanism of V(D)J Recombination from Synaptic RAG1-RAG2 Complex Structures.
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Structural Basis for the Inverted Repeat Preferences of mariner Transposases.
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Solution conformations of early intermediates in Mos1 transposition.