Depressing mitochondria-reticulum interactions protects cardiomyocytes from lethal hypoxia-reoxygenation injury.
Paillard, Melanie; Tubbs, Emily; Thiebaut, Pierre-Alain; Gomez, Ludovic; Fauconnier, Jeremy; Da Silva, Claire Crola; Teixeira, Geoffrey; Mewton, Nathan; Belaidi, Elise; Durand, Annie; Abrial, Maryline; Lacampagne, Alain; Rieusset, Jennifer; Ovize, Michel.
; 128(14): 1555-65, 2013 Oct 01.
Artigo em Inglês | MEDLINE | ID: mdl-23983249
Cyclophilin D Modulates the Cardiac Mitochondrial Target of Isoflurane, Sevoflurane, and Desflurane.
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure.
Resident Macrophages: Near and Dear to Your Heart.
The role of mitochondrial permeability transition in reperfusion-induced cardiomyocyte death depends on the duration of ischemia.
A proliferative burst during preadolescence establishes the final cardiomyocyte number.
Mitochondrial calcium uniporter regulator 1 (MCUR1) regulates the calcium threshold for the mitochondrial permeability transition.
Cardiac T-Tubule Microanatomy and Function.
Physiologic functions of cyclophilin D and the mitochondrial permeability transition pore.
cGMP signals modulate cAMP levels in a compartment-specific manner to regulate catecholamine-dependent signaling in cardiac myocytes.
Adult c-kit(pos) cardiac stem cells are necessary and sufficient for functional cardiac regeneration and repair.