Contribution of CFTR to alveolar fluid clearance by lipoxin A4 via PI3K/Akt pathway in LPS-induced acute lung injury.
Yang, Yi; Cheng, Yang; Lian, Qing-Quan; Yang, Li; Qi, Wei; Wu, De-Rong; Zheng, Xia; Liu, Yong-Jian; Li, Wen-Juan; Jin, Sheng-Wei; Smith, Fang Gao.
; 2013: 862628, 2013.
Artigo em Inglês | MEDLINE | ID: mdl-23766562
Specialized Pro-resolving Mediators Regulate Alveolar Fluid Clearance during Acute Respiratory Distress Syndrome.
Correction of both NBD1 energetics and domain interface is required to restore ΔF508 CFTR folding and function.
Timing of CFTR pore opening and structure of its transition state.
Conformational Changes of CFTR upon Phosphorylation and ATP Binding.
TGF-ß-induced IL-6 prevents development of acute lung injury in influenza A virus-infected F508del CFTR-heterozygous mice.
Atomic Structure of the Cystic Fibrosis Transmembrane Conductance Regulator.
Requirements for efficient correction of ΔF508 CFTR revealed by analyses of evolved sequences.
Human mesenchymal stromal cells reduce influenza A H5N1-associated acute lung injury in vitro and in vivo.
Functional architecture of the CFTR chloride channel.
STRUCTURE, GATING, AND REGULATION OF THE CFTR ANION CHANNEL.