Procoagulant activity induced by vascular injury determines contribution of elevated factor VIII to thrombosis and thrombus stability in mice.
Machlus, Kellie R; Lin, Feng-Chang; Wolberg, Alisa S.
; 118(14): 3960-8, 2011 Oct 06.
Artigo em Inglês | MEDLINE | ID: mdl-21828144
Platelet IκB kinase-ß deficiency increases mouse arterial neointima formation via delayed glycoprotein Ibα shedding.
Novel Paracrine Functions of Smooth Muscle Cells in Supporting Endothelial Regeneration Following Arterial Injury.
Effect of hormone replacement therapy in matrix metalloproteinase expression and intimal hyperplasia development after vascular injury.
The induction of yes-associated protein expression after arterial injury is crucial for smooth muscle phenotypic modulation and neointima formation.
Bone marrow-derived Kruppel-like factor 10 controls reendothelialization in response to arterial injury.
Inhibitory effects of mesenchymal stem cells in intimal hyperplasia after balloon angioplasty.
The interferon stimulated gene 12 inactivates vasculoprotective functions of NR4A nuclear receptors.
HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4.
Palmitoylethanolamide and Polydatin combination reduces inflammation and oxidative stress in vascular injury.
Heparan sulfate side chains have a critical role in the inhibitory effects of perlecan on vascular smooth muscle cell response to arterial injury.