Stat4 is critical for the balance between Th17 cells and regulatory T cells in colitis.
Xu, Jiangnan; Yang, Yu; Qiu, Guixing; Lal, Girdhari; Yin, Na; Wu, Zhihong; Bromberg, Jonathan S; Ding, Yaozhong.
; 186(11): 6597-606, 2011 Jun 01.
Artigo em Inglês | MEDLINE | ID: mdl-21525389
Adipose-Resident Group 1 Innate Lymphoid Cells Promote Obesity-Associated Insulin Resistance.
Expression and regulation of intergenic long noncoding RNAs during T cell development and differentiation.
Signal transducer and activator of transcription 4 is required for the transcription factor T-bet to promote T helper 1 cell-fate determination.
T-Bet Is Dependent on Stat-4 Inhibiting Acute Colitis but Not Stat-1 Using L4 Somatic Antigen of <i>Heligmosomoides polygyrus</i>.
STAT4 Regulates the CD8<sup>+</sup> Regulatory T Cell/T Follicular Helper Cell Axis and Promotes Atherogenesis in Insulin-Resistant <i>Ldlr<sup>-/-</sup></i> Mice.
Epigenetic instability of cytokine and transcription factor gene loci underlies plasticity of the T helper 17 cell lineage.
Opposing roles of STAT4 and Dnmt3a in Th1 gene regulation.
Deletion of a conserved cis-element in the Ifng locus highlights the role of acute histone acetylation in modulating inducible gene transcription.
Discrete roles of STAT4 and STAT6 transcription factors in tuning epigenetic modifications and transcription during T helper cell differentiation.
Genome-wide association study identifies TH1 pathway genes associated with lung function in asthmatic patients.