Microarray analysis of the mammalian thromboxane receptor-Trypanosoma cruzi interaction.
Tanowitz, Herbert B; Mukhopadhyay, Aparna; Ashton, Anthony W; Lisanti, Michael P; Machado, Fabiana S; Weiss, Louis M; Mukherjee, Shankar.
; 10(7): 1132-43, 2011 Apr 01.
Artigo em Inglês | MEDLINE | ID: mdl-21364319
Modulation of host central carbon metabolism and in situ glucose uptake by intracellular Trypanosoma cruzi amastigotes.
Thromboxane A2 is a key regulator of pathogenesis during Trypanosoma cruzi infection.
Trypanosoma cruzi Produces the Specialized Proresolving Mediators Resolvin D1, Resolvin D5, and Resolvin E2.
ROS and Trypanosoma cruzi: Fuel to infection, poison to the heart.
Host triacylglycerols shape the lipidome of intracellular trypanosomes and modulate their growth.
Predominance of Th1 response, increase of megakaryocytes and Kupffer cells are related to survival in Trypanosoma cruzi infected mice treated with Lycopodium clavatum.
Dynamic flux of microvesicles modulate parasite-host cell interaction of Trypanosoma cruzi in eukaryotic cells.
[Costs of Chagas' disease screening test in blood donors in two Colombian blood banks, 2015].
The exocyst is required for trypanosome invasion and the repair of mechanical plasma membrane wounds.
Alterations in pancreatic ß cell function and Trypanosoma cruzi infection: evidence from human and animal studies.