Listerial invasion protein internalin B promotes entry into ileal Peyer's patches in vivo.
Chiba, Sayuri; Nagai, Takeshi; Hayashi, Toshiyuki; Baba, Yukiko; Nagai, Shigenori; Koyasu, Shigeo.
; 55(2): 123-9, 2011 Feb.
Artigo em Inglês | MEDLINE | ID: mdl-21204945
Genomic and phenotypic diversity of Listeria monocytogenes clonal complexes associated with human listeriosis.
Listeria monocytogenes impairs SUMOylation for efficient infection.
Listeria hijacks host mitophagy through a novel mitophagy receptor to evade killing.
ActA of Listeria monocytogenes and Its Manifold Activities as an Important Listerial Virulence Factor.
The Arsenic Resistance-Associated Listeria Genomic Island LGI2 Exhibits Sequence and Integration Site Diversity and a Propensity for Three Listeria monocytogenes Clones with Enhanced Virulence.
Listeria monocytogenes CadC Regulates Cadmium Efflux and Fine-tunes Lipoprotein Localization to Escape the Host Immune Response and Promote Infection.
Metabolic Genetic Screens Reveal Multidimensional Regulation of Virulence Gene Expression in Listeria monocytogenes and an Aminopeptidase That Is Critical for PrfA Protein Activation.
Peyer's Patches as a Portal for DNA Delivery by Lactococcus lactis in Vivo.
CD8α(+) dendritic cells are an obligate cellular entry point for productive infection by Listeria monocytogenes.
The role of Listeria monocytogenes cell wall surface anchor protein LapB in virulence, adherence, and intracellular replication.