Toxoplasma gondii infection confers resistance against BimS-induced apoptosis by preventing the activation and mitochondrial targeting of pro-apoptotic Bax.

Hippe, Diana; Weber, Arnim; Zhou, Liying; Chang, Donald C; Häcker, Georg; Lüder, Carsten G K.
J Cell Sci; 122(Pt 19): 3511-21, 2009 Oct 01.
Artigo em Inglês | MEDLINE | ID: mdl-19737817

Documentos relacionados

  1. A Genome-wide CRISPR Screen in Toxoplasma Identifies Essential Apicomplexan Genes.
  2. Host lipid droplets: An important source of lipids salvaged by the intracellular parasite Toxoplasma gondii.
  3. A unique dynamin-related protein is essential for mitochondrial fission in Toxoplasma gondii.
  4. Toxoplasma effector MAF1 mediates recruitment of host mitochondria and impacts the host response.
  5. Toxoplasma gondii and Neospora caninum induce different host cell responses at proteome-wide phosphorylation events; a step forward for uncovering the biological differences between these closely related parasites.
  6. mRNA pseudouridylation affects RNA metabolism in the parasite <i>Toxoplasma gondii</i>.
  7. Genetic Mapping of Pathogenesis Determinants in Toxoplasma gondii.
  8. An ortholog of Plasmodium falciparum chloroquine resistance transporter (PfCRT) plays a key role in maintaining the integrity of the endolysosomal system in Toxoplasma gondii to facilitate host invasion.
  9. Lytic Cycle of Toxoplasma gondii: 15 Years Later.
  10. Anti-apoptotic effects of SERPIN B3 and B4 via STAT6 activation in macrophages after infection with Toxoplasma gondii.