A fundamental role for interleukin-21 in the generation of T follicular helper cells.
Vogelzang, Alexis; McGuire, Helen M; Yu, Di; Sprent, Jonathan; Mackay, Charles R; King, Cecile.
; 29(1): 127-37, 2008 Jul 18.
Artigo em Inglês | MEDLINE | ID: mdl-18602282
Structural and energetic mechanisms of cooperative autoinhibition and activation of Vav1.
[Regulatory effect of Vav1 on T cells and its relation to clinical diseases].
The guanine nucleotide exchange factor Vav3 regulates differentiation of progenitor cells in the developing mouse retina.
Quantitative proteomics analysis of signalosome dynamics in primary T cells identifies the surface receptor CD6 as a Lat adaptor-independent TCR signaling hub.
The transcription factor GATA3 is critical for the development of all IL-7Rα-expressing innate lymphoid cells.
Vav1 regulates MHCII expression in murine resting and activated B cells.
Cardiac lymphatics are heterogeneous in origin and respond to injury.
A key regulatory role for Vav1 in controlling lipopolysaccharide endotoxemia via macrophage-derived IL-6.
Vav1 is essential for mechanotactic crawling and migration of neutrophils out of the inflamed microvasculature.
Involvement of the guanine nucleotide exchange factor Vav3 in central nervous system development and plasticity.