Increased neurogenesis in young transgenic mice overexpressing human APP(Sw, Ind).
López-Toledano, Miguel A; Shelanski, Michael L.
J Alzheimers Dis
; 12(3): 229-40, 2007 Nov.
Artigo em Inglês | MEDLINE | ID: mdl-18057556
APP mouse models for Alzheimer's disease preclinical studies.
Sorting Out Presenilins in Alzheimer's Disease.
Alzheimer mechanisms and therapeutic strategies.
Alzheimer's-Causing Mutations Shift Aß Length by Destabilizing Î³-Secretase-Aßn Interactions.
Iron-export ferroxidase activity of ß-amyloid precursor protein is inhibited by zinc in Alzheimer's disease.
Directed conversion of Alzheimer's disease patient skin fibroblasts into functional neurons.
ApoE2, ApoE3, and ApoE4 Differentially Stimulate APP Transcription and Aß Secretion.
Loss of clusterin shifts amyloid deposition to the cerebrovasculature via disruption of perivascular drainage pathways.
Amyloid precursor protein and endosomal-lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease.
Amyloid precursor protein processing and Alzheimer's disease.