Human C5aR knock-in mice facilitate the production and assessment of anti-inflammatory monoclonal antibodies.
Lee, Hyun; Zahra, David; Vogelzang, Alexis; Newton, Rebecca; Thatcher, Jenny; Quan, Annie; So, Trina; Zwirner, Jörg; Koentgen, Frank; Padkjaer, Søren B; Mackay, Fabienne; Whitfeld, Peter L; Mackay, Charles R.
; 24(10): 1279-84, 2006 Oct.
Artigo em Inglês | MEDLINE | ID: mdl-16980974
Characterisation of receptor binding by the chemotaxis inhibitory protein of Staphylococcus aureus and the effects of the host immune response.
Characterization of Inhibitors and Monoclonal Antibodies That Modulate the Interaction between Plasmodium falciparum Adhesin PfRh4 with Its Erythrocyte Receptor Complement Receptor 1.
The receptor for complement component C3a mediates protection from intestinal ischemia-reperfusion injuries by inhibiting neutrophil mobilization.
Nuclear Pore Permeabilization Is a Convergent Signaling Event in Effector-Triggered Immunity.
Rad50-CARD9 interactions link cytosolic DNA sensing to IL-1ß production.
Complementary therapies for inflammation.
Membrane protein Crry maintains homeostasis of the complement system.
Quantitative proteomics analysis of signalosome dynamics in primary T cells identifies the surface receptor CD6 as a Lat adaptor-independent TCR signaling hub.
GIMAP1 Is Essential for the Survival of Naive and Activated B Cells In Vivo.
Mast cell anaphylatoxin receptor expression can enhance IgE-dependent skin inflammation in mice.