Reprogramming of CTLs into natural killer-like cells in celiac disease.
Meresse, Bertrand; Curran, Shane A; Ciszewski, Cezary; Orbelyan, Gerasim; Setty, Mala; Bhagat, Govind; Lee, Leanne; Tretiakova, Maria; Semrad, Carol; Kistner, Emily; Winchester, Robert J; Braud, Veronique; Lanier, Lewis L; Geraghty, Daniel E; Green, Peter H; Guandalini, Stefano; Jabri, Bana.
J Exp Med
; 203(5): 1343-55, 2006 May 15.
Artigo em Inglês | MEDLINE | ID: mdl-16682498
Integration of genetic and immunological insights into a model of celiac disease pathogenesis.
Refractory coeliac disease.
Interleukin-15-Dependent T-Cell-like Innate Intraepithelial Lymphocytes Develop in the Intestine and Transform into Lymphomas in Celiac Disease.
Les complications de la maladie cÅliaque.
Persistent small bowel mucosal villous atrophy without symptoms in coeliac disease.
Mucosal healing and risk for lymphoproliferative malignancy in celiac disease: a population-based cohort study.
Is a gluten-free diet necessary in Marsh I intestinal lesions in patients with HLADQ2, DQ8 genotype and without gastrointestinal symptoms?
Recasting the Tissue-Resident Lymphocyte in Celiac Disease.
[Successes and unsolved problems in the study of celiac disease].