c-fos gene expression is induced in a subpopulation of striatal neurons following a single administration of a dopamine D1-receptor agonist in adult rats lesioned with 6-OHDA as neonates.
Soghomonian, J J; Laprade, N; Sandstrom, M; Bruno, J P.
Brain Res Mol Brain Res
; 57(1): 155-60, 1998 Jun 01.
Artigo em Inglês | MEDLINE | ID: mdl-9630593
Upregulation and axonal transport of synaptotagmin-IV in the direct-pathway medium spiny neurons in hemi-parkinsonian rats induced by dopamine D1 receptor stimulation.
Detection of molecular alterations in methamphetamine-activated Fos-expressing neurons from a single rat dorsal striatum using fluorescence-activated cell sorting (FACS).
Changes in CArG-binding protein A expression levels following injection(s) of the D1-dopamine agonist SKF-82958 in the intact and 6-hydroxydopamine-lesioned rat.
D1 Antagonists and D2 Agonists Have Opposite Effects on the Metabolism of Dopamine in the Rat Striatum.
The atypical dopamine D1 receptor agonist SKF 83959 induces striatal Fos expression in rats.
Striatal neurones have a specific ability to respond to phasic dopamine release.
The dopamine D1 receptor agonist, but not the D2 receptor agonist, induces gene expression of Homer 1a in rat striatum and nucleus accumbens.
Neurochemical and behavioural indices of exercise reward are independent of exercise controllability.
N-methyl-D-aspartate (NMDA) receptor composition modulates dendritic spine morphology in striatal medium spiny neurons.
The selective inhibition of the D1 dopamine receptor results in an increase of metabolized dopamine in the rat striatum.