Chronological aging is associated with biophysical and chemical changes in the capsule of Cryptococcus neoformans.
Cordero, Radames J B; Pontes, Bruno; Guimarães, Allan J; Martinez, Luis R; Rivera, Johanna; Fries, Bettina C; Nimrichter, Leonardo; Rodrigues, Marcio L; Viana, Nathan B; Casadevall, Arturo.
; 79(12): 4990-5000, 2011 Dec.
Artigo em Inglês | MEDLINE | ID: mdl-21968999
Titan cells confer protection from phagocytosis in Cryptococcus neoformans infections.
Virulence mechanisms and Cryptococcus neoformans pathogenesis.
Immunity to Cryptococcus neoformans and C. gattii during cryptococcosis.
A Single Protein S-acyl Transferase Acts through Diverse Substrates to Determine Cryptococcal Morphology, Stress Tolerance, and Pathogenic Outcome.
Cryptococcus neoformans can form titan-like cells in vitro in response to multiple signals.
Cryptococcal phospholipase B1 is required for intracellular proliferation and control of titan cell morphology during macrophage infection.
Antibody binding to Cryptococcus neoformans impairs budding by altering capsular mechanical properties.
Isolation and characterization of senescent Cryptococcus neoformans and implications for phenotypic switching and pathogenesis in chronic cryptococcosis.
The Membrane Phospholipid Binding Protein Annexin A2 Promotes Phagocytosis and Nonlytic Exocytosis of Cryptococcus neoformans and Impacts Survival in Fungal Infection.
Variability in innate host immune responses to cryptococcosis.