Modulation of lipid droplets by Mycobacterium leprae in Schwann cells: a putative mechanism for host lipid acquisition and bacterial survival in phagosomes.
Mattos, Katherine A; Lara, Flavio A; Oliveira, Viviane G C; Rodrigues, Luciana S; D'Avila, Heloisa; Melo, Rossana C N; Manso, Pedro P A; Sarno, Euzenir N; Bozza, Patricia T; Pessolani, Maria Cristina V.
; 13(2): 259-73, 2011 Feb.
Artigo em Inglês | MEDLINE | ID: mdl-20955239
The Essential Role of Cholesterol Metabolism in the Intracellular Survival of Mycobacterium leprae Is Not Coupled to Central Carbon Metabolism and Energy Production.
Subversion of Schwann Cell Glucose Metabolism by Mycobacterium leprae.
Identification of functional candidates amongst hypothetical proteins of Mycobacterium leprae Br4923, a causative agent of leprosy.
A Macrophage Response to Mycobacterium leprae Phenolic Glycolipid Initiates Nerve Damage in Leprosy.
Interaction of Mycobacterium leprae with human airway epithelial cells: adherence, entry, survival, and identification of potential adhesins by surface proteome analysis.
Myelination key factor krox-20 is downregulated in Schwann cells and murine sciatic nerves infected by Mycobacterium leprae.
Pathogenic mechanisms of intracellular bacteria.
Do leprosy and tuberculosis generate a systemic inflammatory shift? Setting the ground for a new dialogue between experimental immunology and bioarchaeology.
Leprosy lipid provides the key to Schwann cell entry.
Involvement of 9-O-Acetyl GD3 ganglioside in Mycobacterium leprae infection of Schwann cells.