Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.
Dai, Dao-Fu; Santana, Luis F; Vermulst, Marc; Tomazela, Daniela M; Emond, Mary J; MacCoss, Michael J; Gollahon, Katherine; Martin, George M; Loeb, Lawrence A; Ladiges, Warren C; Rabinovitch, Peter S.
; 119(21): 2789-97, 2009 Jun 02.
Artigo em Inglês | MEDLINE | ID: mdl-19451351
Increased mitochondrial nanotunneling activity, induced by calcium imbalance, affects intermitochondrial matrix exchanges.
Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity.
Cardiac Response to Oxidative Stress Induced by Mitochondrial Dysfunction.
Age-dependent microRNA control of synaptic plasticity in 22q11 deletion syndrome and schizophrenia.
[Specific molecular and morphological changes in cardiomyocytes of hibernating ground squirrels in different periods of annual cycle].
Sarcalumenin alleviates stress-induced cardiac dysfunction by improving Ca2+ handling of the sarcoplasmic reticulum.
Mitochondrial structure and function are not different between nonfailing donor and end-stage failing human hearts.
Postnatal development of mouse heart: formation of energetic microdomains.
Secretory products of guinea pig epicardial fat induce insulin resistance and impair primary adult rat cardiomyocyte function.
Kissing and nanotunneling mediate intermitochondrial communication in the heart.