Human C5aR knock-in mice facilitate the production and assessment of anti-inflammatory monoclonal antibodies.
Lee, Hyun; Zahra, David; Vogelzang, Alexis; Newton, Rebecca; Thatcher, Jenny; Quan, Annie; So, Trina; Zwirner, Jörg; Koentgen, Frank; Padkjaer, Søren B; Mackay, Fabienne; Whitfeld, Peter L; Mackay, Charles R.
; 24(10): 1279-84, 2006 Oct.
Artigo em Inglês | MEDLINE | ID: mdl-16980974
Characterisation of receptor binding by the chemotaxis inhibitory protein of Staphylococcus aureus and the effects of the host immune response.
The receptor for complement component C3a mediates protection from intestinal ischemia-reperfusion injuries by inhibiting neutrophil mobilization.
Mast cell anaphylatoxin receptor expression can enhance IgE-dependent skin inflammation in mice.
Membrane protein Crry maintains homeostasis of the complement system.
Characterization of Inhibitors and Monoclonal Antibodies That Modulate the Interaction between Plasmodium falciparum Adhesin PfRh4 with Its Erythrocyte Receptor Complement Receptor 1.
Complementary therapies for inflammation.
Complement receptor 1 is the host erythrocyte receptor for Plasmodium falciparum PfRh4 invasion ligand.
Rad50-CARD9 interactions link cytosolic DNA sensing to IL-1ß production.
Complement deficiency promotes cutaneous wound healing in mice.
Complement and its receptors: new insights into human disease.